Acid Reflux Medication Interferes with Vitamin and Mineral Absorption

The AHA! Team — Wed, 03 Sep 2025 05:15:21 +0000

Lori Alton via NaturalHealth365 – Gastroesophageal reflux disease, or acid reflux as it is often known, is one of the most common health complaints among adults. So much so that medications to treat the symptoms make up the third highest-selling class of drugs in the United States. It is estimated that a staggering 20% of the the U.S. population suffer with symptoms of acid reflux. While proton-pump inhibiting (PPI) drugs, such as Prilosec, Prevacid, and Nexium, bring welcomed relief from the symptoms of acid reflux, habitual use of these pharmaceuticals over time can be a health risk, increasing the likelihood of nutritional deficiencies, bone fractures, and heart disease. Understanding the cause of acid reflux Acid reflux is caused by the incomplete closure of the sphincter valve – located between the stomach and the esophagus. When this occurs, harsh stomach contents can splash back up across the delicate lining of the esophagus and the throat, nose, and other vulnerable tissues. Most commonly, acid reflux can cause pain in the center of the chest and, at times, an acidic taste in the mouth. While uncomfortable, the true concern is that – left untreated – symptoms of acid reflux will lead to esophageal cancer and other related changes down the road. But one thing should be made perfectly clear: toxic medications will never cure this problem. The dangers of PPI drugs and acid reflux What many acid reflux sufferers don’t realize is that medications to treat their heartburn and other symptoms come at a hefty price. There are Prilosec side effects, for example, that may go unnoticed for months or even years. Using PPIs to gain some control over symptoms may seem helpful, but these medications don’t stop the physical reflux of digestive enzymes, bile, and corrosive food/drinks flowing into the delicate esophageal lining. Continued exposure to the acids and protein-digesting enzymes in stomach fluid can bring about precancerous changes, such as the development of the condition known as Barrett’s esophagus. Reducing stomach acid is also likely to interfere with your body’s ability to extract vitamin B12 from foods. Research shows that most oral acid-suppressing medications, including PPIs, decrease the absorption of vitamin B12 from foods. In one study, 75 percent of PPI users were deficient in vitamin B12, compared with just 11 percent among non-users. Gut problems can be life-threatening Regular use of PPIs may also lead to low blood magnesium levels. Symptoms include fatigue, unsteadiness, numbness/tingling, seizures, and heart rhythm disturbances. In severe cases, hospitalization may be necessary. However, magnesium levels can return to normal when PPI medications are halted. PPI use can also lead to poor calcium absorption, increasing the risk of bone fractures. These medications are also associated with elevated risk for cardiovascular disease and even risk of death due to heart attack. PPIs inhibit the normal production of nitric oxide, a signaling molecule vital for normal blood vessel function. Lower your risk of disease with natural solutions Avoiding the use of PPIs in favor of natural solutions to acid reflux and heartburn results in fewer unwanted impacts on your health. Often, dietary and lifestyle changes hold the key to diminishing occurrence. Avoid processed and “rich” foods, particularly desserts. Do not overeat, and chew your food – very well. In terms of a plant remedy, check out this article about how slippery elm can help you. If you are a smoker, acid reflux is yet another reason to quit. If you still have acid reflux symptoms, consult a trusted holistic physician with experience in natural remedies – including essential oils and herbs – to help guide you toward a full recovery. Sources for this article include: NIH.gov Lifeextension.com To read the original article click here.

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Over-the-Counter Acid Reflux Drug Appears to Improve the Odds of Survival for COVID-19 Patients

AHA Publisher — Mon, 23 Aug 2021 07:00:11 +0000

University of Virginia School of Engineering and Applied Science via News Medical – In the early days of the COVID-19 pandemic, doctors in Wuhan noticed something surprising. Many of the elderly patients who survived the virus were poor: not exactly the demographic you would expect to fare well in a health crisis. A review of the survivors’ medical records revealed that a significant number suffered from chronic heartburn and were taking an inexpensive drug called famotidine, the key ingredient in Pepcid. (Wealthier patients tended to take the more costly drug omeprazole, found in Prilosec.) Was an over-the-counter acid suppressant helping people survive COVID-19? This is how many medical studies begin, said biomedical engineering professor Phil Bourne, who serves as founding dean for the School of Data Science. “There’s often a phenomenon that doctors report anecdotally, or that’s mentioned in passing in a particular research paper, and that provides a clue -; a hook,” he said. Typically, to find out whether a drug is effective in treating a particular medical condition, scientists develop prospective clinical trials. But this method is expensive and can take years, Bourne pointed out. When faced with a global pandemic, it’s helpful to explore other options. That’s where data scientists come in. Bourne and UVA senior scientist Cameron Mura worked with an international team of researchers to analyze information from a database that holds the medical records of millions of COVID-19 patients living in 30 different countries. The team winnowed that number down to around 22,000 people, the largest sample size for a study on famotidine and the disease to date. “The power of the electronic health record, which is really yet to be fully realized as a research tool, is that you’ve suddenly got all this data you can mine to see whether what you determined in passing or anecdotally has any basis.” Phil Bourne, Biomedical Engineering Professor The team’s analysis, which appeared in the journal Signal Transduction & Targeted Therapy(from the Nature publishing group), showed that the data supported findings from other smaller-scale studies. When delivered at high doses (the equivalent of about 10 Pepcid tablets), famotidine appears to improve the odds of survival for COVID-19 patients, especially when it is combined with aspirin. It also seems to hinder the severity of disease progression, making patients less likely to reach the point where they require intubation or a ventilator. The next challenge was to figure out why. Data scientists like Mura and Bourne perform extensive detective work for medical analyses like this one, looking at existing information and drawing upon biochemical and molecular principles to propose a cohesive theory that helps elucidate the population-scale patterns they identify. Mura calls this “weaving a story” based on the data. He needed to work backwards from massive groups of people and draw some possible conclusions about what was happening at a totally different scale -; the scale of proteins that are “one millionth the size of an ant,” he said. One of the most dangerous phenomena COVID-19 can trigger in your body is something called a cytokine storm, which is a potentially fatal amplification of an immune response. When you become sick, your immune system releases inflammatory proteins called cytokines that tell your immune cells how to fight the infection. But in more severe illnesses, cytokine production can spiral out of control, becoming dysregulated. “Basically, your immune system goes haywire and starts attacking things like your otherwise healthy lung tissue because it’s so desperate to kill off the invading virus,” Mura said. “Your own physiology essentially uses a sledgehammer against the pathogen when a fly swatter would suffice.” The team’s theory is that famotidine suppresses that reaction. Although it was developed with a specific purpose in mind -; blocking the histamine receptors that help produce acid in your stomach -; famotidine, like all other medications, can cause side effects. Mura and his colleagues believe that interfering with cytokine storms might be one of them. “It may well be a case of famotidine having a beneficial off-target effect,” Mura said. We generally think of side effects as a bad thing, but in some cases, they can be harnessed to treat other conditions. In the future, it’s possible that famotidine could be re-purposed in this way. But the team’s findings are far from conclusive. Other studies have offered conflicting pictures of what famotidine can do for COVID-19 patients: Some have found that it has a neutral effect and one has even suggested that it might be detrimental. Mura, Bourne and their colleagues recently published a review of existing research on the subject, along with suggestions for a framework that could help reconcile the contradictory reports. Still, with its unique focus on combining famotidine with aspirin and its impressively large sample size, the team’s study has shed further light on an inexpensive and safe potential treatment that would be easy for doctors to prescribe. In the midst of an international health crisis, the study has also laid important groundwork for further research. “Scientific studies are sometimes viewed as the end-all, be-all, but they’re really just a starting point or a springboard,” Mura said. “Any good study raises more questions than it answers, and data science is often what kick-starts that process.” To read the original article click here.

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Acid Reflux Medication Interferes with Vitamin and Mineral Absorption

Over-the-Counter Acid Reflux Drug Appears to Improve the Odds of Survival for COVID-19 Patients